Cancer-Associated Fibroblasts
"Cancer-Associated Fibroblasts" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Subpopulation of heterogeneous fibroblasts within the TUMOR MICROENVIRONMENT that support NEOPLASTIC CELL TRANSFORMATION and NEOPLASTIC PROCESSES.
Descriptor ID |
D000072645
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MeSH Number(s) |
A11.329.228.105
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Concept/Terms |
Cancer-Associated Fibroblasts- Cancer-Associated Fibroblasts
- Cancer-Associated Fibroblast
- Fibroblast, Cancer-Associated
- Fibroblasts, Cancer-Associated
- Cancer Associated Fibroblasts
- Cancer Associated Fibroblast
- Fibroblast, Cancer Associated
- Fibroblasts, Cancer Associated
- Tumor-Associated Fibroblasts
- Fibroblast, Tumor-Associated
- Fibroblasts, Tumor-Associated
- Tumor Associated Fibroblasts
- Tumor-Associated Fibroblast
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Below are MeSH descriptors whose meaning is more general than "Cancer-Associated Fibroblasts".
Below are MeSH descriptors whose meaning is more specific than "Cancer-Associated Fibroblasts".
This graph shows the total number of publications written about "Cancer-Associated Fibroblasts" by people in this website by year, and whether "Cancer-Associated Fibroblasts" was a major or minor topic of these publications.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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2016 | 1 | 0 | 1 |
2017 | 2 | 0 | 2 |
2018 | 2 | 1 | 3 |
2019 | 1 | 3 | 4 |
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Below are the most recent publications written about "Cancer-Associated Fibroblasts" by people in Profiles.
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In silico analysis identifies neuropilin-1 as a potential therapeutic target for SARS-Cov-2 infected lung cancer patients. Aging (Albany NY). 2021 06 24; 13(12):15770-15784.
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mTORC1 hyperactivation in lymphangioleiomyomatosis leads to ACE2 upregulation in type II pneumocytes: implications for COVID-19. Eur Respir J. 2021 02; 57(2).
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The Nutritional Cytokine Leptin Promotes NSCLC by Activating the PI3K/AKT and MAPK/ERK Pathways in NSCLC Cells in a Paracrine Manner. Biomed Res Int. 2019; 2019:2585743.
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The immune milieu of cholangiocarcinoma: From molecular pathogenesis to precision medicine. J Autoimmun. 2019 06; 100:17-26.
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Oxidized ATM-mediated glycolysis enhancement in breast cancer-associated fibroblasts contributes to tumor invasion through lactate as metabolic coupling. EBioMedicine. 2019 Mar; 41:370-383.
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MACC1 overexpression in carcinoma-associated fibroblasts induces the invasion of lung adenocarcinoma cells via paracrine signaling. Int J Oncol. 2019 04; 54(4):1367-1375.
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Cancer-associated fibroblast (CAF)-derived IL32 promotes breast cancer cell invasion and metastasis via integrin ß3-p38 MAPK signalling. Cancer Lett. 2019 02 01; 442:320-332.
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Activation of Farnesoid X Receptor impairs the tumor-promoting function of breast cancer-associated fibroblasts. Cancer Lett. 2018 11 28; 437:89-99.
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Single-Cell Transcriptome Analyses Reveal Endothelial Cell Heterogeneity in Tumors and Changes following Antiangiogenic Treatment. Cancer Res. 2018 05 01; 78(9):2370-2382.
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FOXQ1/NDRG1 axis exacerbates hepatocellular carcinoma initiation via enhancing crosstalk between fibroblasts and tumor cells. Cancer Lett. 2018 03 28; 417:21-34.