"Synoviocytes" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cells on the luminal surface of the SYNOVIAL MEMBRANE. Type A synoviocytes are MACROPHAGES responsible for waste removal from the joint cavity. Fibroblast-like type B synoviocytes are involved in production of joint matrix constituents (e.g., HYALURONAN; COLLAGEN; and FIBRONECTIN).
Descriptor ID |
D000070918
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MeSH Number(s) |
A11.329.835
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Concept/Terms |
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Below are MeSH descriptors whose meaning is more general than "Synoviocytes".
Below are MeSH descriptors whose meaning is more specific than "Synoviocytes".
This graph shows the total number of publications written about "Synoviocytes" by people in this website by year, and whether "Synoviocytes" was a major or minor topic of these publications.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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2017 | 1 | 1 | 2 |
2018 | 6 | 1 | 7 |
2019 | 1 | 2 | 3 |
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Below are the most recent publications written about "Synoviocytes" by people in Profiles.
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TNF-a/calreticulin dual signaling induced NLRP3 inflammasome activation associated with HuR nucleocytoplasmic shuttling in rheumatoid arthritis. Inflamm Res. 2019 Jul; 68(7):597-611.
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Exenatide ameliorates inflammatory response in human rheumatoid arthritis fibroblast-like synoviocytes. IUBMB Life. 2019 07; 71(7):969-977.
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Curculigoside exerts significant anti-arthritic effects in vivo and in vitro via regulation of the JAK/STAT/NF-?B signaling pathway. Mol Med Rep. 2019 Mar; 19(3):2057-2064.
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A20 inhibits the release of inflammatory cytokines by suppressing the activation of the nuclear factor-kappa B pathway in osteoarthritic fibroblast-like synoviocytes. Biochem Biophys Res Commun. 2019 01 15; 508(3):877-881.
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Control of cytokine mRNA degradation by the histone deacetylase inhibitor ITF2357 in rheumatoid arthritis fibroblast-like synoviocytes: beyond transcriptional regulation. Arthritis Res Ther. 2018 07 20; 20(1):148.
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Follistatin-like protein 1 induction of matrix metalloproteinase 1, 3 and 13 gene expression in rheumatoid arthritis synoviocytes requires MAPK, JAK/STAT3 and NF-?B pathways. J Cell Physiol. 2018 01; 234(1):454-463.
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Identification and suppression of epidermal growth factor receptor variant III signaling in fibroblast-like synoviocytes from aggressive rheumatoid arthritis by the mimotope. Immunol Lett. 2018 06; 198:74-80.
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Bcl-XL and Mcl-1 upregulation by calreticulin promotes apoptosis resistance of fibroblast-like synoviocytes via activation of PI3K/Akt and STAT3 pathways in rheumatoid arthritis. Clin Exp Rheumatol. 2018 Sep-Oct; 36(5):841-849.
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Long Non-Coding RNA GAPLINC Promotes Tumor-Like Biologic Behaviors of Fibroblast-Like Synoviocytes as MicroRNA Sponging in Rheumatoid Arthritis Patients. Front Immunol. 2018; 9:702.
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Towards an arthritis flare-responsive drug delivery system. Nat Commun. 2018 04 03; 9(1):1275.