"Receptor, Melatonin, MT2" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A melatonin receptor subtype primarily found expressed in the BRAIN and RETINA.
| Descriptor ID |
D044123
|
| MeSH Number(s) |
D12.776.543.750.695.440.750
|
| Concept/Terms |
Receptor, Melatonin, MT2- Receptor, Melatonin, MT2
- Melatonin 1b Receptor
- Receptor, Melatonin 1b
- MT2 Receptor
- Receptor, MT2
- Mel 1b Receptor
- Melatonin MT2 Receptor
- MT2 Receptor, Melatonin
- Receptor, Melatonin MT2
|
Below are MeSH descriptors whose meaning is more general than "Receptor, Melatonin, MT2".
Below are MeSH descriptors whose meaning is more specific than "Receptor, Melatonin, MT2".
This graph shows the total number of publications written about "Receptor, Melatonin, MT2" by people in this website by year, and whether "Receptor, Melatonin, MT2" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2010 | 0 | 1 | 1 |
| 2017 | 1 | 0 | 1 |
| 2018 | 1 | 2 | 3 |
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Below are the most recent publications written about "Receptor, Melatonin, MT2" by people in Profiles.
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Does the melatonin receptor 1B gene polymorphism have a role in postoperative delirium? PLoS One. 2018; 13(11):e0207941.
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Genetic polymorphisms in candidate genes are not associated with increased vincristine-related peripheral neuropathy in Arab children treated for acute childhood leukemia: a single institution study. Pharmacogenet Genomics. 2018 08; 28(8):189-195.
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Inhibiting MT2-TFE3-dependent autophagy enhances melatonin-induced apoptosis in tongue squamous cell carcinoma. J Pineal Res. 2018 Mar; 64(2).
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Analysis of MTNR1B gene polymorphisms in relationship with IRS2 gene variants, epicardial fat thickness, glucose homeostasis and cognitive performance in the elderly. Chronobiol Int. 2017; 34(8):1083-1093.
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The inhibition of apoptosis by melatonin in VSC4.1 motoneurons exposed to oxidative stress, glutamate excitotoxicity, or TNF-alpha toxicity involves membrane melatonin receptors. J Pineal Res. 2010 Mar; 48(2):157-69.