Transcription Factor RelA
"Transcription Factor RelA" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A subunit of NF-kappa B that is primarily responsible for its transactivation function. It contains a C-terminal transactivation domain and an N-terminal domain with homology to PROTO-ONCOGENE PROTEINS C-REL.
| Descriptor ID |
D051996
|
| MeSH Number(s) |
D12.776.260.600.249 D12.776.660.600.249 D12.776.930.600.249
|
| Concept/Terms |
Transcription Factor RelA- Transcription Factor RelA
- RelA, Transcription Factor
- NF-kappa B p65
- NF kappa B p65
- p65, NF-kappa B
- NF-kappa B p65 Subunit
- NF kappa B p65 Subunit
- Transcription Factor p65
- p65, Transcription Factor
- p65 Transcription Factor
- Transcription Factor, p65
- v-Rel Reticuloendotheliosis Viral Oncogene Homolog A
- v Rel Reticuloendotheliosis Viral Oncogene Homolog A
- NF-kappaB 65-kDa
- NF kappaB 65 kDa
|
Below are MeSH descriptors whose meaning is more general than "Transcription Factor RelA".
Below are MeSH descriptors whose meaning is more specific than "Transcription Factor RelA".
This graph shows the total number of publications written about "Transcription Factor RelA" by people in this website by year, and whether "Transcription Factor RelA" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1998 | 0 | 1 | 1 |
| 2001 | 0 | 1 | 1 |
| 2004 | 0 | 1 | 1 |
| 2008 | 1 | 0 | 1 |
| 2011 | 1 | 0 | 1 |
| 2012 | 0 | 1 | 1 |
| 2013 | 0 | 1 | 1 |
| 2015 | 0 | 1 | 1 |
| 2016 | 2 | 0 | 2 |
| 2017 | 3 | 9 | 12 |
| 2018 | 9 | 14 | 23 |
| 2019 | 1 | 4 | 5 |
| 2021 | 0 | 1 | 1 |
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Below are the most recent publications written about "Transcription Factor RelA" by people in Profiles.
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PCA-based unsupervised feature extraction for gene expression analysis of COVID-19 patients. Sci Rep. 2021 08 30; 11(1):17351.
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Ergosterol peroxide exhibits antiviral and immunomodulatory abilities against porcine deltacoronavirus (PDCoV) via suppression of NF-?B and p38/MAPK signaling pathways in vitro. Int Immunopharmacol. 2021 Apr; 93:107317.
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MDA5 Governs the Innate Immune Response to SARS-CoV-2 in Lung Epithelial Cells. Cell Rep. 2021 01 12; 34(2):108628.
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Investigating the mechanism of ShuFeng JieDu capsule for the treatment of novel Coronavirus pneumonia (COVID-19) based on network pharmacology. Int J Med Sci. 2020; 17(16):2511-2530.
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Epithelial cell-specific loss of function of Miz1 causes a spontaneous COPD-like phenotype and up-regulates Ace2 expression in mice. Sci Adv. 2020 08; 6(33):eabb7238.
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Identifying crucial genes for prognosis in septic patients: Gene integration study based on PRISMA guidelines. Medicine (Baltimore). 2019 Aug; 98(33):e16807.
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High-Dose Paraquat Induces Human Bronchial 16HBE Cell Death and Aggravates Acute Lung Intoxication in Mice by Regulating Keap1/p65/Nrf2 Signal Pathway. Inflammation. 2019 Apr; 42(2):471-484.
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Boswellic acid has anti-inflammatory effects and enhances the anticancer activities of Temozolomide and Afatinib, an irreversible ErbB family blocker, in human glioblastoma cells. Phytother Res. 2019 06; 33(6):1670-1682.
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Role of TLR-4 in anti-ß2-glycoprotein I-induced activation of peritoneal macrophages and vascular endothelial cells in mice. Mol Med Rep. 2019 May; 19(5):4353-4363.
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Curculigoside exerts significant anti-arthritic effects in vivo and in vitro via regulation of the JAK/STAT/NF-?B signaling pathway. Mol Med Rep. 2019 Mar; 19(3):2057-2064.