"Cellular Senescence" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Process by which cells irreversibly stop dividing and enter a state of permanent growth arrest without undergoing CELL DEATH. Senescence can be induced by DNA DAMAGE or other cellular stresses, such as OXIDATIVE STRESS.
Descriptor ID |
D016922
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MeSH Number(s) |
G04.043
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Concept/Terms |
Cell Aging- Cell Aging
- Cellular Ageing
- Ageing, Cellular
- Aging, Cell
- Senescence, Replicative
- Cellular Aging
- Aging, Cellular
- Replicative Senescence
- Cell Ageing
- Ageing, Cell
Senescence-Associated Secretory Phenotype- Senescence-Associated Secretory Phenotype
- Phenotype, Senescence-Associated Secretory
- Secretory Phenotype, Senescence-Associated
- Senescence Associated Secretory Phenotype
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Below are MeSH descriptors whose meaning is more general than "Cellular Senescence".
Below are MeSH descriptors whose meaning is more specific than "Cellular Senescence".
This graph shows the total number of publications written about "Cellular Senescence" by people in this website by year, and whether "Cellular Senescence" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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1997 | 1 | 0 | 1 |
2003 | 0 | 1 | 1 |
2014 | 1 | 1 | 2 |
2015 | 1 | 0 | 1 |
2016 | 1 | 0 | 1 |
2017 | 13 | 16 | 29 |
2018 | 7 | 9 | 16 |
2019 | 2 | 4 | 6 |
2020 | 0 | 4 | 4 |
2021 | 2 | 0 | 2 |
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Below are the most recent publications written about "Cellular Senescence" by people in Profiles.
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The Mechanisms of the Growth Inhibitory Effects of Paclitaxel on Gefitinib-resistant Non-small Cell Lung Cancer Cells. Cancer Genomics Proteomics. 2021 Sep-Oct; 18(5):661-673.
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Fisetin for COVID-19 in skilled nursing facilities: Senolytic trials in the COVID era. J Am Geriatr Soc. 2021 11; 69(11):3023-3033.
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Cellular senescence in lymphoid organs and immunosenescence. Aging (Albany NY). 2021 08 12; 13(15):19920-19941.
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Characterization of the SARS-CoV-2 Host Response in Primary Human Airway Epithelial Cells from Aged Individuals. Viruses. 2021 08 12; 13(8).
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SARS-CoV-2 Spike Protein Induces Paracrine Senescence and Leukocyte Adhesion in Endothelial Cells. J Virol. 2021 08 10; 95(17):e0079421.
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Improving gene network inference with graph wavelets and making insights about ageing-associated regulatory changes in lungs. Brief Bioinform. 2021 07 20; 22(4).
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The Nexus Between Telomere Length and Lymphocyte Count in Seniors Hospitalized With COVID-19. J Gerontol A Biol Sci Med Sci. 2021 07 13; 76(8):e97-e101.
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NK cells on the ViP stage of COVID-19. EBioMedicine. 2021 Jul; 69:103458.
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Cellular senescence is a potential severity factor for COVID-19: Suitable targets required to eliminate cellular senescence. Respir Med. 2021 09; 186:106517.
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ELV-N32 and RvD6 isomer decrease pro-inflammatory cytokines, senescence programming, ACE2 and SARS-CoV-2-spike protein RBD binding in injured cornea. Sci Rep. 2021 06 17; 11(1):12787.