Endoplasmic Reticulum Stress
"Endoplasmic Reticulum Stress" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Various physiological or molecular disturbances that impair ENDOPLASMIC RETICULUM function. It triggers many responses, including UNFOLDED PROTEIN RESPONSE, which may lead to APOPTOSIS; and AUTOPHAGY.
| Descriptor ID |
D059865
|
| MeSH Number(s) |
G04.434
|
| Concept/Terms |
Endoplasmic Reticulum Stress- Endoplasmic Reticulum Stress
- Endoplasmic Reticulum Stresses
- Reticulum Stress, Endoplasmic
- Reticulum Stresses, Endoplasmic
- Stresses, Endoplasmic Reticulum
- Stress, Endoplasmic Reticulum
|
Below are MeSH descriptors whose meaning is more general than "Endoplasmic Reticulum Stress".
Below are MeSH descriptors whose meaning is more specific than "Endoplasmic Reticulum Stress".
This graph shows the total number of publications written about "Endoplasmic Reticulum Stress" by people in this website by year, and whether "Endoplasmic Reticulum Stress" was a major or minor topic of these publications.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2014 | 1 | 0 | 1 |
| 2016 | 2 | 1 | 3 |
| 2017 | 20 | 18 | 38 |
| 2018 | 11 | 15 | 26 |
| 2019 | 2 | 6 | 8 |
| 2020 | 1 | 0 | 1 |
| 2021 | 0 | 3 | 3 |
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Below are the most recent publications written about "Endoplasmic Reticulum Stress" by people in Profiles.
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Endoplasmic reticulum stress and NF-kB activation in SARS-CoV-2 infected cells and their response to antiviral therapy. IUBMB Life. 2022 01; 74(1):93-100.
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Pneumocytes are distinguished by highly elevated expression of the ER stress biomarker GRP78, a co-receptor for SARS-CoV-2, in COVID-19 autopsies. Cell Stress Chaperones. 2021 09; 26(5):859-868.
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Increased susceptibility of human endothelial cells to infections by SARS-CoV-2 variants. Basic Res Cardiol. 2021 07 05; 116(1):42.
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Role of JAK/STAT in Interstitial Lung Diseases; Molecular and Cellular Mechanisms. Int J Mol Sci. 2021 Jun 09; 22(12).
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Induction of the Proinflammatory Chemokine Interleukin-8 Is Regulated by Integrated Stress Response and AP-1 Family Proteins Activated during Coronavirus Infection. Int J Mol Sci. 2021 May 26; 22(11).
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COVID-19 in Africa: an ovarian victory? J Ovarian Res. 2021 May 21; 14(1):70.
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SARS-CoV-2 suppresses mRNA expression of selenoproteins associated with ferroptosis, endoplasmic reticulum stress and DNA synthesis. Food Chem Toxicol. 2021 Jul; 153:112286.
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Glucose-regulated protein (GRP78) is an important cell surface receptor for viral invasion, cancers, and neurological disorders. IUBMB Life. 2021 06; 73(6):843-854.
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The interplay between the immune system and SARS-CoV-2 in COVID-19 patients. Arch Virol. 2021 Aug; 166(8):2109-2117.
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The Unfolded Protein Response and Autophagy on the Crossroads of Coronaviruses Infections. Front Cell Infect Microbiol. 2021; 11:668034.