Receptors, Transforming Growth Factor beta
"Receptors, Transforming Growth Factor beta" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cell-surface proteins that bind transforming growth factor beta and trigger changes influencing the behavior of cells. Two types of transforming growth factor receptors have been recognized. They differ in affinity for different members of the transforming growth factor beta family and in cellular mechanisms of action.
| Descriptor ID |
D018125
|
| MeSH Number(s) |
D12.776.543.750.705.852.720 D12.776.543.750.750.400.820
|
| Concept/Terms |
Receptors, Transforming Growth Factor beta- Receptors, Transforming Growth Factor beta
- Transforming Growth Factor beta Receptors
- Receptors, TGF-beta
- Receptors, TGF beta
- TGF-beta Receptor
- Receptor, TGF-beta
- TGF beta Receptor
- TGF-beta Receptors
- TGF beta Receptors
- Transforming Growth Factor beta Receptor
|
Below are MeSH descriptors whose meaning is more general than "Receptors, Transforming Growth Factor beta".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Transforming Growth Factor beta".
This graph shows the total number of publications written about "Receptors, Transforming Growth Factor beta" by people in this website by year, and whether "Receptors, Transforming Growth Factor beta" was a major or minor topic of these publications.
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| Year | Major Topic | Minor Topic | Total |
|---|
| 2008 | 1 | 0 | 1 |
| 2012 | 1 | 0 | 1 |
| 2013 | 0 | 1 | 1 |
| 2017 | 4 | 3 | 7 |
| 2018 | 0 | 4 | 4 |
| 2019 | 1 | 1 | 2 |
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Below are the most recent publications written about "Receptors, Transforming Growth Factor beta" by people in Profiles.
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Transforming growth factor beta receptor II (TGFBR2) promoter region polymorphism. Breast Cancer Res Treat. 2019 12; 178(3):713.
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Concomitant Expression of Prolactin Receptor and TGFß Receptors in Breast Cancer: Association with Less Aggressive Phenotype and Favorable Patient Outcome. Int J Mol Sci. 2019 Apr 02; 20(7).
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TGFß1-Smad Signaling Pathway Participates in Interleukin-33 Induced Epithelial-to-Mesenchymal Transition of A549 Cells. Cell Physiol Biochem. 2018; 50(2):757-767.
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Interaction of E3 Ubiquitin Ligase MARCH7 with Long Noncoding RNA MALAT1 and Autophagy-Related Protein ATG7 Promotes Autophagy and Invasion in Ovarian Cancer. Cell Physiol Biochem. 2018; 47(2):654-666.
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Autocrine transforming growth factor-ß/activin A-Smad signaling induces hepatic progenitor cells undergoing partial epithelial-mesenchymal transition states. Biochimie. 2018 May; 148:87-98.
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Differential expression of TGFß isoforms in breast cancer highlights different roles during breast cancer progression. Tumour Biol. 2018 Jan; 40(1):1010428317748254.
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Activation of STAT3 integrates common profibrotic pathways to promote fibroblast activation and tissue fibrosis. Nat Commun. 2017 10 24; 8(1):1130.
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Evaluation of incidence, significance, and prognostic role of circulating tumor microemboli and transforming growth factor-ß receptor I in head and neck cancer. Head Neck. 2017 11; 39(11):2283-2292.
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A late presentation of Loeys-Dietz syndrome associated with an aortic root aneurysm. Ann R Coll Surg Engl. 2017 Mar; 99(3):e114-e115.
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An engineered transforming growth factor ß (TGF-ß) monomer that functions as a dominant negative to block TGF-ß signaling. J Biol Chem. 2017 04 28; 292(17):7173-7188.